Frequently asked questions
Semax is a synthetic heptapeptide derived from adrenocorticotropic hormone (ACTH 4-10) with the addition of a C-terminal Pro-Gly-Pro sequence. It is studied in laboratory and preclinical models for its influence on neuroprotection, cognitive processes, and cellular stress responses. Research has investigated its ability to modulate neurotrophic factor expression, neurotransmitter systems, and oxidative stress pathways.
Semax has been studied in neurological, cognitive, and systemic models, with research highlighting its role in neuroprotection, cognitive function, and stress response. Studies also report activity in neurotransmitter modulation, oxidative stress pathways, and recovery processes in preclinical settings.
Key Areas of Research:
• Neurological: neuroprotection, signaling, pathways
• Cognitive: memory, learning, function
• Systemic: stress response, repair, resilience
Together, these findings suggest broad experimental potential for Semax across neurological, cognitive, and systemic pathways. By influencing neurotransmitter systems and supporting protective and restorative responses, Semax provides a versatile platform for research into cognition, stress regulation, and systemic resilience in laboratory models.
